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Numerous pathological mechanisms, including the accumulation and deposition of A-peptides, the formation of neurofibrillary tangles, DNA oxidation, protein oxidation, and lipid peroxidation, exacerbate oxidative damage to neurons [4]

M.A.M.H.: Conceptualization, investigation, data curation, writingoriginal draft, writingreview and editing

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Interestingly, although lipid peroxidation amplifies PARP-1 activation, inhibiting PARP-1 can significantly reduce lipid peroxidation and maintain mitochondrial integrity, underscoring the link between PARP-1 overactivation and oxidative damage in promoting parthanatos [142, 143]

Grundke-Iqbal I, Iqbal K, Tung YC, Quinlan M, Wisniewski HM, Binder LI