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Although the actual mechanisms of MTX-induced liver and kidney damage are unknown, one of the hypotheses is the breakdown in cellular antioxidant defences, which increases the production of reactive oxygen species and nitrogen species, inhibits cytosolic NADP-dependent dehydrogenase and NADP malic enzyme, lowers glutathione (GSH), superoxide dismutase (SOD) and catalase levels and reduces the effectiveness of the antioxidant defence system protecting the cell against reactive oxygen species (Reference Savran, Cicek and Doguc7)

Lipidomics analysis reveals that peroxisomes promote iron dependent death by synthesizing polyunsaturated ether phospholipids (PUFA-ePLs)

Subsequently, other authors conducted cross checks on the extracted data to maintain its integrity and reliability

Biorheology 24, 589597

The weekly injectable medication is unique because it mimics the hormone amylin, which helps control blood sugar

From Krebs to clinic: glutamine metabolism to cancer therapy