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In fact, adenosine acting through microglial A 2A receptors potentiates nitric oxide release [484], and in PD models, A 2A receptor antagonists reduce neuroinflammation by suppressing microglial activation and pro-inflammatory cytokine production, contributing to neuronal survival

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E.NdubaV.El-BeshlawyA.et al (2019)

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For TMJ patients, it reduces inflammatory activity in the joint capsule and supports the resolution of chronic synovitis

Here are the key ones documented in research: Immunosuppressants (cyclosporine, tacrolimus, azathioprine) Glutathione may modulate immune response, potentially counteracting these drugs