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Glutamine exits astrocytes via the bidirectional N system transporters, SNAT3 and SNAT5 [21], and enter neurons via the unidirectional system A transporters, SNAT1, SNAT2 [21], and SNAT7 [22]
They work in (slightly) different ways Zepbound and Ozempic also have distinct active ingredients that work in slightly different ways Ozempic targets GLP-1 receptors, while Zepbound targets both GLP-1 and GIP receptors
Altogether, these results indicate that pharmacological inhibition of MAOB with KDS2010 effectively mitigates PTSD-associated deficits by reducing aberrant astrocytic GABA, restoring behavioral functions, and supporting the therapeutic potential of MAOB inhibitors in PTSD treatment
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