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Similar to dNK cells, miR-483-3p inhibitor-transfected NK cells derived from UCB/CD34 + HSCs demonstrated upregulation of perforin and CD107a (Supplementary Fig

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Oxidative stress is present in the early stages of CKD, contributing to renal function deterioration with increased antioxidant consumption and ROS generation [38, 39]

In this study, we demonstrated that the correction of glycine deficiency with dietary supplements increased the urinary excretion of acylglycine derived from the intermediates of the BCAAs (isobutyrylglycine, tigylglycine, and isovalerylglycine) and fatty acid oxidation (hexanoylglycine)