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2014), resulting in an increased expression of 18-kDa translator protein, associated with the inflammatory activation of CNS (Chen and Guilarte 2008)

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[5]Seneviratne, Uthpala

the classic view is that the adrenal glands are structurally normal, and there is no evidence of adrenal insufficiency or Addisons disease, which is an autoimmune disease, meaning that our own antibodies attack the gland damaging its structure and function

Contrarily, combinational effect of Al + SeNP-MT 1&2 was more

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