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Our in vitro and in vivo models reveal that GPR68 expression inversely correlates with ferroptosis markers (ACSL4, SLC7A11, GPX4) during OGD/R and spinal I/R
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Prior analyses have either used models that exclude the results of cardiovascular outcomes trials (with any reductions in cardiovascular and kidney outcomes being extrapolated only from changes in HbA 1c [13]), considered only specific high-risk populations [45, 46], or modelled the populations used in cardiovascular outcomes trials, which represent only 2060% of all people with type 2 diabetes [14]
Fibrin as an Inducer of Fibrosis in the Tunica Albuginea of the Rat: A New Animal Model of Peyronies Disease
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