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FNDC3B protects steatosis and ferroptosis via the AMPK pathway in alcoholic fatty liver disease

The deposition of GAG was detected by alcian blue staining

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These results suggest that reduced homocysteine causes protein thiol homocysteinylation that may induce smooth muscle cell proliferation by binding to a redox receptor and that may exacerbate atherosclerotic lesion formation by inducing collagen expression (Tyagi, 1998)

GSH levels decreased after 6 h in E100 and K1E100

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