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This cellular damage activates hepatic stellate cells (HSCs), which are key players in liver fibrogenesis, contributing to the deposition of extracellular matrix proteins and the progression of fibrosis [8,9,10]

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Once iron metabolism is disordered, Fe stored in aforementioned locations is released, and the iron pool is converted into a labile iron pool, leading to excessive Fe content in the cytoplasm [53]

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