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Deficiency can cause nerve pain and heightened sensitivity

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The SH groups are potential sites of reversible oxidative modification by S-glutathiolation and S-nitrosylation but they are also susceptible to irreversible damage by oxidative conditions

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The triple-agonist mechanism (GLP-1/GIP/glucagon) appears to offer incremental benefit over dual-agonist (tirzepatide) or single-agonist (semaglutide) approaches