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Recombinant CXCL10IL18R1 interface peptide injection rescued the NRTP-induced decrease in ovarian function The above results demonstrated that CXC10 antibody injection rescued against ovarian damage

Tumor cells are especially vulnerable to ROS-based therapies due to their limited antioxidant defenses

Glutathione acts as a protective shield, taking the damage and saving the surrounding cell components from oxidation

This is a real, but heavily managed/monitored issue

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An increase in ROS production or a decrease in ROS-scavenging capacity due to exogenous stimuli or endogenous metabolic alterations can disrupt redox homeostasis, lead to an overall increase intracellular ROS levels, or oxidative stress [29,30]