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Therapeutic potential of isorhamnetin following acetaminophen-induced hepatotoxicity through targeting NLRP3/NF-B/Nrf2

1 Introduction Graves disease (GD), an autoimmune thyroid disease (AITD), is the most prevalent cause of hyperthyroidism, potentially resulting in goiter, protruding eyeballs, pretibial mucous edema, and an elevated metabolic state, all of which pose significant health risks (1, 2)