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2.1.2 Pharmacodynamics of curcumin Curcumin executes its therapeutic benefits through several molecular mechanisms and has thus shown its pharmacodynamic versatility

Proinflammatory cytokines TNF- and IL1- activate the canonical NF-B pathway that induce the gene expression of cyclooxigenase-2, inducible nitric oxide synthase-3, inflammatory cytokines, and matrix metalloproteinases [75] These changes lead to the damage of TJs, decreased endothelial integrity and increased permeability at the BBB [73, 76], and to neuroinflammation and the development of chronic neurodegenerative diseases such as AD [10, 77]

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This mechanism is particularly relevant for men with circulatory-related ED, often classified as arteriogenic ED