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In the extracellular environment, EBs can infect neighboring cells and continue propagating (Saka and Valdivia, Chlamydia encounter non bacteriocidal stress conditions, like those arising from exposure to certain antibiotics, cytokines or nutrient deprivation, the bacteria respond by markedly reducing RBs division and production of infectious particles, being able to rapidly resume to normal replication once the stressing conditions are removed (Figure 1A)

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