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This is achieved by stimulating the activity of immune cells, such as M1 macrophages and CD8+ T lymphocytes, and by promoting the secretion of pro-inflammatory cytokines, including interferon-gamma

Problems can occur when inflammatory signaling becomes persistent, poorly regulated, or disconnected from the original trigger

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Taurine ameliorated homocysteine-induced H9C2 cardiomyocyte apoptosis by modulating endoplasmic reticulum stress

Research by Lee et al

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